Activation Of Proteinase 3 Contributes To Nonalcoholic Fatty Liver Disease And Insulin Resistance
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The Role of Intestinal C‐type Regenerating Islet Derived‐3
increasing epidemic. Nonalcoholic fatty liver disease (NAFLD) has a prevalence of >70% in adults who are obese. Disease progression to nonalco-holic steatohepatitis (NASH), ﬁbrosis, cirrhosis, and ﬁnally hepatocellular carcinoma worsens the prognosis, and therefore NASH is on the rise to become the leading indication for liver transplantation.
Hematopoietic Tissue Factor-Protease-Activated Receptor 2
liver disease (NAFLD), a disease that ranges from fatty liver to the more severe condition of steatohepatitis, which can, if leftunchecked, progress to ﬁbrosis and cirrhosis in upto 25%
Activation of Proteinase 3 Contributes to nonalcoholic Fatty
Activation of inflammatory pathways is known to accompany development of obesity-induced nonalcoholic fatty liver disease (NAFLD), insulin resistance and type 2 diabetes. In addition to caspase-1, the neutrophil serine proteases proteinase 3, neutro-